GDNF Protects against Aluminum-Induced Apoptosis in Rabbits by Upregulating Bcl-2 and Bcl-XL and Inhibiting Mitochondrial Bax Translocation
- 1 October 2001
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 8 (5), 764-773
- https://doi.org/10.1006/nbdi.2001.0429
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- RETRACTED: Cyclosporin A inhibits Al-induced cytochrome c release from mitochondria in aged rabbitsJournal of Alzheimer's Disease, 2001
- Bcl-2 Family Gene Products in Cerebral Ischemia and Traumatic Brain InjuryJournal of Neurotrauma, 2000
- Mitochondria: execution centralFEBS Letters, 2000
- Apoptotic crosstalk between the endoplasmic reticulum and mitochondria controlled by Bcl-2Oncogene, 2000
- Involvement of Caspase‐3‐Like Protease in the Mechanism of Cell Death Following Focally Evoked Limbic SeizuresJournal of Neurochemistry, 2000
- Apoptosis and neurologic diseaseThe American Journal of Medicine, 2000
- The parkinsonian neurotoxin MPP+ opens the mitochondrial permeability transition pore and releases cytochrome c in isolated mitochondria via an oxidative mechanismBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1999
- Bax-induced Cytochrome C Release from Mitochondria Is Independent of the Permeability Transition Pore but Highly Dependent on Mg2+ IonsThe Journal of cell biology, 1998
- Glutamate regulation of GDNF gene expression in the striatum and primary striatal astrocytesNeuroReport, 1995
- Regulation of Lymphocyte Survival by the BCL-2 Gene FamilyAnnual Review of Immunology, 1995