Relationship between tolerance to GABAA agonist and bursting properties in neocortical neurons during GABA-withdrawal syndrome
- 1 October 1989
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 498 (2), 289-298
- https://doi.org/10.1016/0006-8993(89)91107-4
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Ca2+-mediated suppression of the GABA-response through modulation of chloride channel gating in frog sensory neuronesNeuroscience Letters, 1988
- GABAergic inhibition and the induction of spontaneous epileptiform activity by low chloride and high potassium in the hippocampal sliceBrain Research, 1988
- The GABA-withdrawal syndrome: a new model of focal epileptogenesisBrain Research, 1988
- Electrophysiology of GABAA and GABAB receptor subtypesTrends in Neurosciences, 1988
- Anticonvulsant effect of intracortical, chronic infusion of GABA in kindled rats: Focal seizures upon withdrawalExperimental Neurology, 1987
- Bursting in human epileptogenic neocortex is depressed by an antagonistNeuroscience Letters, 1987
- Epileptogenic γ-aminobutyric acid-withdrawal syndrome after chronic, intracortical infusion in baboonsNeuroscience Letters, 1987
- Nipecotic acid, an uptake blocker, prevents fading of the γ-aminobutyric acid effectBrain Research, 1986
- Electrophysiological properties of neocortical neurons in vitroJournal of Neurophysiology, 1982
- Genesis of epileptic interictal spikes. New knowledge of cortical feedback systems suggests a neurophysiological explanation of brief paroxysmsBrain Research, 1973