Retinoid-regulated expression of BCL-2 and tissue transglutaminase during the differentiation and apoptosis of human myeloid leukemia (HL-60) cells
- 30 June 1996
- journal article
- Published by Elsevier in Leukemia Research
- Vol. 20 (6), 499-505
- https://doi.org/10.1016/0145-2126(95)00118-2
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Activation of Retinoid X Receptors Induces Apoptosis in HL-60 Cell LinesMolecular and Cellular Biology, 1995
- Dysregulated bcl-2 expression inhibits apoptosis but not differentiation of retinoic acid-induced HL-60 granulocytesBlood, 1994
- Bcl2 inhibits apoptosis associated with terminal differentiation of HL- 60 myeloid leukemia cellsBlood, 1994
- Synthesis of high specific activity tritium-labeled [3H]-9-cis-retinoic acid and its application for identifying retinoids with unusual binding propertiesJournal of Medicinal Chemistry, 1994
- Bcl-2 and the regulation of programmed cell deathThe Journal of cell biology, 1994
- Bcl-2 oncoprotein blocks chemotherapy-induced apoptosis in a human leukemia cell lineBlood, 1993
- Retinoids Selective for Retinoid X Receptor Response PathwaysScience, 1992
- 9-cis retinoic acid is a high affinity ligand for the retinoid X receptorCell, 1992
- 9-Cis retinoic acid stereoisomer binds and activates the nuclear receptor RXRαNature, 1992
- HL-60 cells induced to differentiate towards neutrophils subsequently die via apoptosisClinical and Experimental Immunology, 1990