BCL-2 Transduction, Using a Herpes Simplex Virus Amplicon, Protects Hippocampal Neurons from Transient Global Ischemia
- 1 March 1999
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 156 (1), 130-137
- https://doi.org/10.1006/exnr.1998.7004
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Bcl-x1 Bax Interaction after Transient Global IschemiaJournal of Cerebral Blood Flow & Metabolism, 1998
- Development of an HSV-Based Vector for the Treatment of Parkinson's DiseaseExperimental Neurology, 1997
- Engineering herpes simplex virus vectors for gene transfer to neuronsNature Medicine, 1997
- HSV-1 vector mediated transfer of BDNF into cerebellar granule cellsNeuroReport, 1996
- Expression of the Apoptosis‐Effector Gene, Bax, Is Up‐Regulated in Vulnerable Hippocampal CA1 Neurons Following Global IschemiaJournal of Neurochemistry, 1996
- Ischemic Delayed Neuronal DeathStroke, 1995
- Long-Term Behavioral Recovery in Parkinsonian Rats by an HSV Vector Expressing Tyrosine HydroxylaseScience, 1994
- On the regional distribution of heparan sulfate proteoglycan immunoreactivity in the rat brainBrain Research, 1994
- The proto-oncogene bcl-2 can selectively rescue neurotrophic factor-dependent neurons from apoptosisCell, 1993
- A Reliable Method for Demonstrating Axonal Degeneration Shortly After AxotomyStain Technology, 1980