The AMPA Receptor Subunits GluR-A and GluR-B Reciprocally Modulate Spinal Synaptic Plasticity and Inflammatory Pain
Open Access
- 1 November 2004
- Vol. 44 (4), 637-650
- https://doi.org/10.1016/j.neuron.2004.10.029
Abstract
No abstract availableKeywords
This publication has 55 references indexed in Scilit:
- Experience Strengthening Transmission by Driving AMPA Receptors into SynapsesScience, 2003
- AMPA Receptor Trafficking and Synaptic PlasticityAnnual Review of Neuroscience, 2002
- Subunit-Specific Rules Governing AMPA Receptor Trafficking to Synapses in Hippocampal Pyramidal NeuronsCell, 2001
- Neuronal Plasticity: Increasing the Gain in PainScience, 2000
- Glutamate receptor subunit 2-selective antibody shows a differential distribution of calcium-impermeable AMPA receptors among populations of neuronsJournal of Comparative Neurology, 1997
- Thalamic NMDA receptors modulate inflammation-produced hyperalgesia in the ratPain, 1997
- Enhanced LTP in Mice Deficient in the AMPA Receptor GluR2Neuron, 1996
- Basal and touch-evoked fos-like immunoreactivity during experimental inflammation in the ratPain, 1996
- Relative abundance of subunit mRNAs determines gating and Ca2+ permeability of AMPA receptors in principal neurons and interneurons in rat CNSNeuron, 1995
- A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesiaPain, 1988