Prostaglandin Synthesis and Inactivation in Kidneys and Lungs of Rats with Experimental Diabetes

Abstract
Microsomal prostaglandin [PG] synthesis and tissue PG concentrations in the kidneys of rats made diabetic by pretreatment with alloxan or streptozotocin were significantly decreased but were unchanged in lungs. PG inactivation was increased in kidneys of alloxan-diabetic rats but not changed in streptozotocin-diabetic kidneys or in the lungs of either group. These changes in PG metabolism may cause a renal PG deficit and could contribute to certain pathological sequelae observed in diabetes mellitus.

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