Agonist-induced alteration in the membrane form of muscarinic cholinergic receptors.
Open Access
- 1 October 1985
- journal article
- research article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 260 (24), 13060-13066
- https://doi.org/10.1016/s0021-9258(17)38838-5
Abstract
No abstract availableThis publication has 29 references indexed in Scilit:
- Pertussis toxin does not inhibit muscarinic-receptor-mediated phosphoinositide hydrolysis or calcium mobilizationBiochemical Journal, 1985
- Role of guanine nucleotide binding protein in the activation of polyphosphoinositide phosphodiesteraseNature, 1985
- Inositol trisphosphates in carbachol-stimulated rat parotid glandsBiochemical Journal, 1984
- Translocation and Uncoupling of theβ-Adrenergic Receptor in Rat Lung after Catecholamine Promoted Desensitizationin VivoEndocrinology, 1984
- Pertussis toxin differentiates between two mechanisms of attenuation of cyclic AMP accumulation by muscarinic cholinergic receptors.Proceedings of the National Academy of Sciences, 1984
- Characterization of an altered membrane form of the beta-adrenergic receptor produced during agonist-induced desensitization.Journal of Biological Chemistry, 1983
- Short‐term disappearance of muscarinic cell surface receptors in carbachol‐induced desensitizationFEBS Letters, 1983
- Catecholamine-Induced Alteration in Sedimentation Behavior of Membrane Bound β-Adrenergic ReceptorsScience, 1980
- Receptor-mediated endocytosis: insights from the lipoprotein receptor system.Proceedings of the National Academy of Sciences, 1979
- Evidence for internalization of the recognition site of β-adrenergic receptors during receptor subsensitivity induced by (-)-isoproterenolProceedings of the National Academy of Sciences, 1979