Bid Is Cleaved by Calpain to an Active Fragment in Vitro and during Myocardial Ischemia/Reperfusion
Open Access
- 1 August 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (33), 30724-30728
- https://doi.org/10.1074/jbc.m103701200
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Decreased expression of high-molecular-weight calmodulin-binding protein and its correlation with apoptosis in ischemia-reperfused rat heartCell Calcium, 2001
- Smac, a Mitochondrial Protein that Promotes Cytochrome c–Dependent Caspase Activation by Eliminating IAP InhibitionCell, 2000
- Calpain and caspase: can you tell the difference?, by Kevin K.W. Wang: Vol. 23, pp. 20–26Trends in Neurosciences, 2000
- Caspase Activation and Mitochondrial Cytochrome C Release during Hypoxia-mediated Apoptosis of Adult Ventricular MyocytesJournal of Molecular and Cellular Cardiology, 2000
- Co-localization of the Cysteine Protease Caspase-3 with Apoptotic Myocytes afterIn VivoMyocardial Ischemia and Reperfusion in the RatJournal of Molecular and Cellular Cardiology, 1998
- Preconditioning rabbit cardiomyocytes: role of pH, vacuolar proton ATPase, and apoptosis.Journal of Clinical Investigation, 1996
- Effect of Ischemic Preconditioning on Mitochondrial Oxidative Phosphorylation and High Energy Phosphates in Rat HeartsJournal of Molecular and Cellular Cardiology, 1996
- Control and kinetic analysis of ischemia-damaged heart mitochondria: which parts of the oxidative phosphorylation system are affected by ischemia?Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1995
- Reperfusion injury induces apoptosis in rabbit cardiomyocytes.Journal of Clinical Investigation, 1994
- Functional behaviour of isolated heart muscle mitochondria after in situ ischemiaBasic Research in Cardiology, 1977