Pathophysiology of sickle cell disease: Role of cellular and genetic modifiers
- 1 October 2001
- journal article
- review article
- Published by Elsevier BV in Seminars in Hematology
- Vol. 38 (4), 299-306
- https://doi.org/10.1016/s0037-1963(01)90023-x
Abstract
No abstract availableKeywords
This publication has 79 references indexed in Scilit:
- FKLF-2: a novel Krüppel-like transcriptional factor that activates globin and other erythroid lineage genesBlood, 2000
- FKLF, a Novel Krüppel-Like Factor That Activates Human Embryonic and Fetal β-Like Globin GenesMolecular and Cellular Biology, 1999
- Pathogenesis and Treatment of Sickle Cell DiseaseNew England Journal of Medicine, 1997
- PATHOPHYSIOLOGY OF SICKLE CELL ANEMIAHematology/Oncology Clinics of North America, 1996
- PATHOPHYSIOLOGY OF VASO-OCCLUSIONHematology/Oncology Clinics of North America, 1996
- Hydroxyurea and Sickle Cell Anemia Clinical Utility of a Myelosuppressive “Switching” AgentMedicine, 1996
- Thrombospondin from activated platelets promotes sickle erythrocyte adherence to human microvascular endothelium under physiologic flow: a potential role for platelet activation in sickle cell vaso-occlusionBlood, 1993
- Evidence for a direct reticulocyte origin of dense red cells in sickle cell anemia.JCI Insight, 1991
- Hemoglobin S polymerization: primary determinant of the hemolytic and clinical severity of the sickling syndromesBlood, 1985
- Irreversibly sickled erythrocytes: a consequence of the heterogeneous distribution of hemoglobin types in sickle-cell anemiaJCI Insight, 1968