A Comparison of Aspirin plus Tirofiban with Aspirin plus Heparin for Unstable Angina

Abstract
Activation of platelets is central to the pathophysiology of unstable angina. We studied whether inhibition of the final common pathway for platelet aggregation with tirofiban, a nonpeptide glycoprotein IIb/IIIa receptor antagonist, would improve clinical outcome in this condition.

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