Severe malarial anemia of low parasite burden in rodent models results from accelerated clearance of uninfected erythrocytes
Open Access
- 1 February 2006
- journal article
- research article
- Published by American Society of Hematology in Blood
- Vol. 107 (3), 1192-1199
- https://doi.org/10.1182/blood-2005-08-3460
Abstract
Severe malarial anemia (SMA) is the most frequent life-threatening complication of malaria and may contribute to the majority of malarial deaths worlKeywords
This publication has 53 references indexed in Scilit:
- Plasmodium falciparum rhoptry protein RSP2 triggers destruction of the erythroid lineageBlood, 2005
- Immunological processes in malaria pathogenesisNature Reviews Immunology, 2005
- The global distribution of clinical episodes of Plasmodium falciparum malariaNature, 2005
- Malarial anaemia: mechanisms and implications of insufficient erythropoiesis during blood-stage malariaInternational Journal for Parasitology, 2004
- Inappropriately low reticulocytosis in severe malarial anemia correlates with suppression in the development of late erythroid precursorsBlood, 2004
- Leishmaniasis host response loci (lmr1–3) modify disease severity through a Th1/Th2-independent pathwayGenes & Immunity, 2003
- Malaria and anemiaCurrent Opinion in Hematology, 2003
- Severe falciparum malariaTransactions of the Royal Society of Tropical Medicine and Hygiene, 2000
- Malaria-a neglected disease?Parasitology, 1992
- Dynamic Alteration in Splenic Function during Acute falciparum MalariaNew England Journal of Medicine, 1987