Differential Molecular Assemblies Underlie the Dual Function of Axin in Modulating the Wnt and JNK Pathways
Open Access
- 1 August 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (34), 32152-32159
- https://doi.org/10.1074/jbc.m104451200
Abstract
No abstract availableKeywords
This publication has 54 references indexed in Scilit:
- Dimerization Choices Control the Ability of Axin and Dishevelled to Activate c-Jun N-terminal Kinase/Stress-activated Protein KinaseJournal of Biological Chemistry, 2000
- Axin-Induced Apoptosis Depends on the Extent of Its JNK Activation and Its Ability to Down-Regulate β-Catenin LevelsBiochemical and Biophysical Research Communications, 2000
- Axin and hepatocellular carcinomasNature Genetics, 2000
- Down-regulation of β-Catenin by the Colorectal Tumor Suppressor APC Requires Association with Axin and β-CateninJournal of Biological Chemistry, 2000
- Diverse functions of JNK signaling and c-Jun in stress response and apoptosisOncogene, 1999
- Regulation of β-Catenin Signaling by the B56 Subunit of Protein Phosphatase 2AScience, 1999
- The oncogenic activation of β-cateninCurrent Opinion in Genetics & Development, 1999
- MECHANISMS OF WNT SIGNALING IN DEVELOPMENTAnnual Review of Cell and Developmental Biology, 1998
- Downregulation of β-catenin by human Axin and its association with the APC tumor suppressor, β-catenin and GSK3βCurrent Biology, 1998
- Functional interaction of β-catenin with the transcription factor LEF-1Nature, 1996