THE ENHANCING EFFECT OF T-2 TOXIN ON DELAYED HYPERSENSITIVITY IN MICE
- 1 January 1977
- journal article
- research article
- Published by Editorial Committee of Japanese Journal of Infectious Diseases, National Institute of Infectious Dis in Japanese Journal of Medical Science and Biology
- Vol. 30 (3), 159-163
- https://doi.org/10.7883/yoken1952.30.159
Abstract
Fusarium solani T-2 toxin, 1 of chemically defined fungal metabolites, enhanced DH [delayed hypersensitivity] response to SRBC [sheep red blood cells] in mice. Effecting timing of toxin administration was different from that of CY [cyclophosphamide]. T-2 toxin seemed to interfere with influences of suppressor cells possibly generated in the DH response.This publication has 4 references indexed in Scilit:
- Genetic control of specific immune suppression. I. Experimental conditions for the stimulation of suppressor cells by the copolymer L-glutamic acid50-L-tyrosine50 (GT) in nonresponder BALB/c mice.The Journal of Experimental Medicine, 1975
- Augmentation of delayed-type hypersensitivity by doses of cyclophosphamide which do not affect antibody responses.The Journal of Experimental Medicine, 1975
- POTENTIATION OF T-CELL-MEDIATED IMMUNITY BY SELECTIVE SUPPRESSION OF ANTIBODY FORMATION WITH CYCLOPHOSPHAMIDEThe Journal of Experimental Medicine, 1974
- CELLULAR AND HUMORAL IMMUNE RESPONSES IN MICEJapanese Journal of Medical Science and Biology, 1973