Cysteine-Rich and Basic Domain HIV-1 Tat Peptides Inhibit Angiogenesis and Induce Endothelial Cell Apoptosis
- 1 May 2001
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 283 (2), 469-479
- https://doi.org/10.1006/bbrc.2001.4790
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Peptides Encoded by Exon 6 of VEGF Inhibit Endothelial Cell Biological Responses and Angiogenesis Induced by VEGFBiochemical and Biophysical Research Communications, 2001
- Vascular endothelial growth factor-induced prostacyclin production is mediated by a protein kinase C (PKC)-dependent activation of extracellular signal-regulated protein kinases 1 and 2 involving PKC-δ and by mobilization of intracellular Ca2+Biochemical Journal, 2001
- Vascular ProtectionArteriosclerosis, Thrombosis, and Vascular Biology, 2000
- VEGF Gene Transfer Reduces Intimal Thickening via Increased Production of Nitric Oxide in Carotid ArteriesHuman Gene Therapy, 1997
- Mechanisms of angiogenesisNature, 1997
- Vascular endothelial growth factorEuropean Journal Of Cancer, 1996
- The Vascular Endothelial Growth Factor Receptor Flt-1 Mediates Biological ActivitiesJournal of Biological Chemistry, 1996
- Synergy between basic fibroblast growth factor and HIV-1 Tat protein in induction of Kaposi's sarcomaNature, 1994
- Tat and the HIV-1 promoterCurrent Opinion in Cell Biology, 1993
- Tat protein of HIV-1 stimulates growth of cells derived from Kaposi's sarcoma lesions of AIDS patientsNature, 1990