Inflammatory pain-induced signaling events following a conditional deletion of the N-methyl-d-aspartate receptor in spinal cord dorsal horn
- 19 June 2008
- journal article
- Published by Elsevier in Neuroscience
- Vol. 155 (3), 948-958
- https://doi.org/10.1016/j.neuroscience.2008.06.024
Abstract
No abstract availableThis publication has 39 references indexed in Scilit:
- Nerve Conduction Blockade in the Sciatic Nerve Prevents but Does Not Reverse the Activation of p38 Mitogen-activated Protein Kinase in Spinal Microglia in the Rat Spared Nerve Injury ModelAnesthesiology, 2007
- Synergistic Control of Protein Kinase Cγ Activity by Ionotropic and Metabotropic Glutamate Receptor Inputs in Hippocampal NeuronsJournal of Neuroscience, 2006
- An Antisense Oligonucleotide to the N-Methyl-d-aspartate (NMDA) Subunit NMDAR1 Attenuates NMDA-Induced Nociception, Hyperalgesia, and Morphine ToleranceJournal of Pharmacology and Experimental Therapeutics, 2004
- MAPK activation in nociceptive neurons and pain hypersensitivityLife Sciences, 2004
- Protein Kinase C ϵ and γ: Involvement in Formalin-Induced Nociception in Neonatal RatsJournal of Pharmacology and Experimental Therapeutics, 2004
- Pain: Molecular mechanismsThe Journal of Pain, 2000
- Neuronal Plasticity: Increasing the Gain in PainScience, 2000
- Immunohistochemical localization of insulin-like growth factor binding protein-5 in the developing rat nervous systemDevelopmental Brain Research, 1996
- Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures.Journal of Histochemistry & Cytochemistry, 1981
- Efficient Analysis of Experimental ObservationsAnnual Review of Pharmacology and Toxicology, 1980