IGF I Induction of p53 Requires Activation of MAP Kinase in Cardiac Muscle Cells
- 28 April 1998
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 245 (3), 912-917
- https://doi.org/10.1006/bbrc.1998.8540
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Coordinated effects of insulin-like growth factor I on inhibitory pathways of cell cycle progression in cultured cardiac muscle cells.Endocrinology, 1995
- Binding of insulin-like growth factors (IGF-I and IGF-II) to the IGF-II/mannose 6-phosphate receptor in fetal rat myocardium.Endocrinology, 1994
- Induction of myocardial insulin-like growth factor-I gene expression in left ventricular hypertrophy.Circulation, 1994
- The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinasesCell, 1993
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- Improvement of doxorubicin induced cardiomyopathy in rats treated with insulin-like growth factor ICardiovascular Research, 1993
- A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasiaCell, 1992
- Multiple components in an epidermal growth factor-stimulated protein kinase cascadeJournal of Biological Chemistry, 1991
- Cellular Pattern of Insulin-Like Growth Factor-I (IGF-I) and Type I IGF Receptor Gene Expression in Early Organogenesis: Comparison with IGF-II Gene ExpressionMolecular Endocrinology, 1990
- Requirement for integration of signals from two distinct phosphorylation pathways for activation of MAP kinaseNature, 1990