AhR- and ERK-dependent pathways function synergistically to mediate 2,3,7,8-tetrachlorodibenzo-p-dioxin suppression of peroxisome proliferator-activated receptor-γ1 expression and subsequent adipocyte differentiation
- 15 May 2003
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 189 (1), 11-27
- https://doi.org/10.1016/s0041-008x(03)00083-8
Abstract
No abstract availableKeywords
This publication has 65 references indexed in Scilit:
- DNA Synthesis and Mitotic Clonal Expansion Is Not a Required Step for 3T3-L1 Preadipocyte Differentiation into AdipocytesJournal of Biological Chemistry, 2001
- Molecular Regulation of AdipogenesisAnnual Review of Cell and Developmental Biology, 2000
- Activation of Peroxisome Proliferator-activated Receptor γ Bypasses the Function of the Retinoblastoma Protein in Adipocyte DifferentiationJournal of Biological Chemistry, 1999
- PPARγ induces cell cycle withdrawal: inhibition of E2F/DP DNA-binding activity via down-regulation of PP2AGenes & Development, 1997
- CCAAT/Enhancer‐Binding Protein β is not Affected by Tetrachlorodibenzo‐p‐dioxin (TCDD) Inhibition of 3T3‐L1 Preadipocyte DifferentiationObesity Research, 1997
- Inhibition of Adipogenesis Through MAP Kinase-Mediated Phosphorylation of PPARγScience, 1996
- Adipocyte Differentiation: When precursors are also regulatorsCurrent Biology, 1995
- EGF triggers neuronal differentiation of PC12 cells that overexpress the EGF receptorCurrent Biology, 1994
- Multiparameter flow cytometric analysis of the effects of indomethacin on adipocyte differentiation in A31T6 cellsCell Proliferation, 1993
- Is a serotonergic mechanism involved in 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced appetite suppression in the Sprague-Dawley rat?Archives of Toxicology, 1991