Persistent Down-Regulation of Fli1, a Suppressor of Collagen Transcription, in Fibrotic Scleroderma Skin
Open Access
- 31 August 2003
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 163 (2), 571-581
- https://doi.org/10.1016/s0002-9440(10)63685-1
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Scleroderma and Smads: Dysfunctional Smad family dynamics culminating in fibrosisArthritis & Rheumatism, 2002
- Ets1 Is an Effector of the Transforming Growth Factor β (TGF-β) Signaling Pathway and an Antagonist of the Profibrotic Effects of TGF-βJournal of Biological Chemistry, 2002
- Molecular aspects of sclerodermaFrontiers in Bioscience-Landmark, 2002
- The role of Fli-1 in normal cell function and malignant transformationOncogene, 2000
- Ets transcription factors cooperate with Sp1 to activate the human Tenascin-C promoterOncogene, 1999
- A potential role for protease nexin 1 overexpression in the pathogenesis of sclerodermaJCI Insight, 1999
- Transcriptional Regulation of the Human α2(I) Collagen GenePublished by Elsevier ,1996
- Characterization of a GC-rich Region Containing Sp1 Binding Site(s) as a Constitutive Responsive Element of the α2(I) Collagen Gene in Human FibroblastsJournal of Biological Chemistry, 1995
- Direct demonstration of transcriptional activation of collagen gene expression in systemic sclerosis fibroblasts: Insensitivity to TGFβ1 stimulationBiochemical and Biophysical Research Communications, 1992
- Expression of Intercellular Adhesion Molecule-1 (ICAM-1) in the Skin of Patients with Systemic SclerodermaJournal of Investigative Dermatology, 1991