Cardiac hypertrophy: from fetal to fatal?
- 1 August 1989
- journal article
- review article
- Published by Wiley in Clinical Physiology and Functional Imaging
- Vol. 9 (4), 315-320
- https://doi.org/10.1111/j.1475-097x.1989.tb00985.x
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- The Creatine Kinase System in Normal and Diseased Human MyocardiumNew England Journal of Medicine, 1985
- Redistribution of creatine kinase isoenzymes in chronically overloaded myocardiumCardiovascular Research, 1985
- Effects of pressure overload, left ventricular hypertrophy on beta-adrenergic receptors, and responsiveness to catecholamines.Journal of Clinical Investigation, 1984
- Training-induced increase in myofibrillar ATPase intermediate fibers in human skeletal muscleMuscle & Nerve, 1982
- Decreased Catecholamine Sensitivity and β-Adrenergic-Receptor Density in Failing Human HeartsNew England Journal of Medicine, 1982
- Isoenzyme pattern and activity of myocardial creatine phosphokinase under heart adaptation to prolonged overloadBasic Research in Cardiology, 1982
- Alterations in cardiac oxygen consumption under chronic pressure overloadBasic Research in Cardiology, 1982
- Structural differences in the heavy chains of rat ventricular myosin isoenzymesFEBS Letters, 1979
- Changes in muscle fibre type distribution in man after physical training: A sign of fibre type transformation?Acta Physiologica Scandinavica, 1978
- Biochemical "Defect" in the Hypertrophied and Failing HeartCirculation, 1973