Wnt signaling function in Alzheimer’s disease
- 1 August 2000
- journal article
- review article
- Published by Elsevier in Brain Research Reviews
- Vol. 33 (1), 1-12
- https://doi.org/10.1016/s0165-0173(00)00021-7
Abstract
No abstract availableKeywords
This publication has 143 references indexed in Scilit:
- Lithium protects cultured neurons against β‐amyloid‐induced neurodegenerationFEBS Letters, 1999
- Neurotoxicity of acetylcholinesterase amyloid β‐peptide aggregates is dependent on the type of Aβ peptide and the AChE concentration present in the complexesFEBS Letters, 1999
- Tau is a candidate gene for chromosome 17 frontotemporal dementiaAnnals of Neurology, 1998
- Presenilin 1 Binds to Amyloid Precursor Protein DirectlyBiochemical and Biophysical Research Communications, 1997
- Acetylcholinesterase promotes the aggregation of amyloid-β-peptide fragments by forming a complex with the growing fibrils 1 1Edited by A. R. FershtJournal of Molecular Biology, 1997
- Lithium inhibits Alzheimer's disease‐like tau protein phosphorylation in neuronsFEBS Letters, 1997
- Overexpression of the mouse dishevelled‐1 protein inhibits GSK‐3β‐mediated phosphorylation of tau in transfected mammalian cellsFEBS Letters, 1997
- Changes of p53 in the Brains of Patients with Alzheimer's DiseaseBiochemical and Biophysical Research Communications, 1997
- Extracellular matrix regulates the amount of the β-amyloid precursor protein and its amyloidogenic fragmentsJournal of Cellular Physiology, 1996
- Neuropathological stageing of Alzheimer-related changesActa Neuropathologica, 1991