Different host-cell shutoff strategies related to the matrix protein lead to persistence of vesicular stomatitis virus mutants on fibroblast cells
- 22 May 2001
- journal article
- Published by Elsevier in Virus Research
- Vol. 76 (1), 87-102
- https://doi.org/10.1016/s0168-1702(01)00251-9
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Identification of a Consensus Mutation in M Protein of Vesicular Stomatitis Virus from Persistently Infected Cells That Affects Inhibition of Host-Directed Gene ExpressionVirology, 1997
- Activity of Vesicular Stomatitis Virus M Protein Mutants in Cell Rounding Is Correlated with the Ability to Inhibit Host Gene Expression and Is Not Correlated with Virus Assembly FunctionVirology, 1997
- Induction of apoptotic DNA fragmentation by the infection of vesicular stomatitis virusVirus Research, 1995
- Quantitation of RNA using the polymerase chain reactionTrends in Genetics, 1993
- Use of an Aqueous Soluble Tetrazolium/Formazan Assay for Cell Growth Assays in CultureCancer Communications, 1991
- Genetic evidence for multiple functions of the matrix protein of vesicular stomatitis virusJournal of General Virology, 1990
- The isolation of interferon-inducing mutants of vesicular stomatitis virus with altered viral P function for the inhibition of total protein synthesisVirology, 1987
- Isolation of biologically active ribonucleic acid from sources enriched in ribonucleaseBiochemistry, 1979
- Persistent Noncytocidal Vesicular Stomatitis Virus Infections Mediated by Defective T Particles that Suppress Virion TranscriptaseProceedings of the National Academy of Sciences, 1974
- Etude Genetique du Virus de la Stomatite Vesiculaire: Classement de Mutants Thermosensibles Spontanes en Groupes de ComplementationJournal of General Virology, 1970