Involvement of the contact phase and intrinsic pathway in herpes simplex virus-initiated plasma coagulation
- 1 May 2010
- journal article
- Published by Elsevier in Journal of Thrombosis and Haemostasis
- Vol. 8 (5), 1037-1043
- https://doi.org/10.1111/j.1538-7836.2010.03789.x
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Factor XI contributes to thrombin generation in the absence of factor XIIBlood, 2009
- Contributions of contact activation pathways of coagulation factor XII in plasmaJournal of Biomedical Materials Research Part A, 2008
- A Cell-Based Model of Thrombin GenerationSeminars in Thrombosis and Hemostasis, 2006
- Mathematical modeling of material-induced blood plasma coagulationBiomaterials, 2006
- Herpes simplex virus type 1-encoded glycoprotein C contributes to direct coagulation Factor X–virus bindingBiochemical Journal, 2005
- Herpes simplex virus type 1-encoded glycoprotein C enhances coagulation factor VIIa activity on the virusThrombosis and Haemostasis, 2004
- Quantitation of viral load in neonatal herpes simplex virus infection and comparison between type 1 and type 2Journal of Medical Virology, 2002
- Infection and AtherosclerosisArteriosclerosis, Thrombosis, and Vascular Biology, 2000
- Association between Prior Cytomegalovirus Infection and the Risk of Restenosis after Coronary AtherectomyNew England Journal of Medicine, 1996
- Glycoprotein C of herpes simplex virus 1 acts as a receptor for the C3b complement component on infected cellsNature, 1984