Nitric Oxide Inhibits c-Jun N-Terminal Kinase 2 (JNK2) via S-Nitrosylation
- 1 June 1998
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 247 (3), 809-813
- https://doi.org/10.1006/bbrc.1998.8788
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Nitric Oxide Modulates the c-Jun N-terminal Kinase/Stress-Activated Protein Kinase Activity through Activating c-Jun N-Terminal Kinase KinaseBiochemistry, 1997
- Effects of Redox-Related Congeners of NO on Apoptosis and Caspase-3 ActivityNitric Oxide, 1997
- Size Control: The Regulation of Cell Numbers in Animal DevelopmentCell, 1996
- Polynitrosylated proteins: characterization, bioactivity, and functional consequences.Proceedings of the National Academy of Sciences, 1996
- Splenic B lymphocyte programmed cell death is prevented by nitric oxide release through mechanisms involving sustained Bcl-2 levels.Journal of Clinical Investigation, 1995
- The nitric oxide donors, azide and hydroxylamine, inhibit the programmed cell death of cytokine‐deprived human eosinophilsFEBS Letters, 1995
- Reversible Inactivation of Calpain Isoforms by Nitric OxideBiochemical and Biophysical Research Communications, 1995
- Nitric oxide produced by human B lymphocytes inhibits apoptosis and Epstein-Barr virus reactivationCell, 1994
- JNK1: A protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domainCell, 1994
- Probing the Structure and Mechanism of Ras Protein with an Expanded Genetic CodeScience, 1993