Mechanism of membrane depolarization caused by the Alzheimer Aβ1–42 peptide
- 17 May 2002
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 293 (4), 1197-1203
- https://doi.org/10.1016/s0006-291x(02)00346-7
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- Clearing the Brain's Amyloid CobwebsNeuron, 2001
- Mechanism and prevention of neurotoxicity caused by β-amyloid peptides: relation to Alzheimer's diseaseBrain Research, 1997
- Compromised mitochondrial function leads to increased cytosolic calcium and to activation of MAP kinasesProceedings of the National Academy of Sciences, 1997
- Attenuation of β‐Amyloid Neurotoxicity In Vitro by Potassium‐Induced DepolarizationJournal of Neurochemistry, 1996
- Structural model for the β-amyloid fibril based on interstrand alignment of an antiparallel-sheet comprising a C-terminal peptideNature Structural & Molecular Biology, 1995
- The toxicity in vitro of β-amyloid proteinBiochemical Journal, 1995
- β-Amyloid precursor protein metabolites and loss of neuronal Ca2+ homeostasis in Alzheimer's diseaseTrends in Neurosciences, 1993
- Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGFBrain Research, 1993
- beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicityJournal of Neuroscience, 1992
- Use of Oxonol V as a probe of membrane potential in proteoliposomes containing cytochrome oxidase in the submitochondrial orientationBiochemistry, 1990