T-Lymphocyte Clones Initiated from Lesional Psoriatic Skin Release Growth Factors that Induce Keratinocyte Proliferation
- 1 November 1993
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 101 (5), 695-700
- https://doi.org/10.1111/1523-1747.ep12371678
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- UM4D4+ (CDw60) T Cells Are Compartmentalized into Psoriatic Skin and Release Lymphokines That Induce a Keratinocyte Phenotype Expressed in Psoriatic LesionsJournal of Investigative Dermatology, 1990
- Mechanisms of Cyclosporine A Inhibition of Antigen-Presenting Activity in Uninvolved and Lesional Psoriatic EpidermisJournal of Investigative Dermatology, 1990
- A Rapid Fluorometric Assay for the Determination of Keratinocyte Proliferation In VitroJournal of Investigative Dermatology, 1989
- Lymphocytes and macrophages of the epidermis and dermis in lesional psoriatic skin, but not epidermal Langerhans cells, are depleted by treatment with cyclosporin AArchives of Dermatological Research, 1989
- Keratinocyte growth regulation by the products of immune cells.The Journal of Experimental Medicine, 1988
- Psoriasis: a disease of abnormal Keratinocyte proliferation induced by T lymphocytesImmunology Today, 1986
- Cyclosporin A inhibits T-cell growth factor gene expression at the level of mRNA transcription.Proceedings of the National Academy of Sciences, 1984
- Evolution, maturation, and regression of lesions of psoriasisThe American Journal of Dermatopathology, 1979
- Disturbance of HL-A Antigen Frequency in PsoriasisNew England Journal of Medicine, 1972
- Continuous culture of human lymphoblasts from peripheral blood of a child with acute leukemiaCancer, 1965