Thyroid Hormones: Control of Terminal Oxidation
- 30 August 1963
- journal article
- research article
- Published by American Association for the Advancement of Science (AAAS) in Science
- Vol. 141 (3583), 816-818
- https://doi.org/10.1126/science.141.3583.816
Abstract
Triiodothyronine administered to thyroidectomized rats preferentially increased the total capacity of the electron transport phosphorylation system when isolated mitochondria from the liver were tested with different substrates, but it caused little increase in the activity of the slowest dehydrogenases and no uncoupling. The increased activity appeared to be partly due to direct activation of some component of the electron transport system, although triiodothyronine injection also stimulated the incorporation of amino acids by isolated mitochondria.Keywords
This publication has 20 references indexed in Scilit:
- The action of thyroid hormones at the cell levelBiochemical Journal, 1963
- The influence of thyroxine on oxidative phosphorylation and related reactionsBiochimica et Biophysica Acta, 1963
- Incorporation of amino acids into the protein of isolated mitochondria. A search for optimum conditions and a relationship to oxidative phosphorylationBiochemical Journal, 1962
- Control of Basal Metabolic Rate by Thyroid Hormones and Cellular FunctionNature, 1962
- Some actions of thyroxine on oxidative phosphorylationBiochimica et Biophysica Acta, 1960
- Effects of physiological levels of thyroxine in vivo on respiration and phosphorylation in rat liver fractionsAmerican Journal of Physiology-Legacy Content, 1959
- The action of thyroxine on mitochondria and oxidative phosphorylationBiochimica et Biophysica Acta, 1955
- RESPIRATORY ENZYMES IN OXIDATIVE PHOSPHORYLATIONJournal of Biological Chemistry, 1955
- The mode of action of thyroxinArchives of Biochemistry and Biophysics, 1951