Mechanisms of NO/cGMP-Dependent Vasorelaxation
- 27 October 2000
- journal article
- other
- Published by Wolters Kluwer Health in Circulation Research
- Vol. 87 (9), 825-830
- https://doi.org/10.1161/01.res.87.9.825
Abstract
—Both cGMP-dependent and -independent mechanisms have been implicated in the regulation of vascular tone by NO. We analyzed acetylcholine (ACh)- and NO-induced relaxation in pressurized sma...Keywords
This publication has 28 references indexed in Scilit:
- Secondary Endothelial Dysfunction: Hypertension and Heart FailureJournal of Molecular and Cellular Cardiology, 1999
- The Alternative: EDHFJournal of Molecular and Cellular Cardiology, 1999
- The Large Conductance, Voltage-dependent, and Calcium-sensitive K+ Channel, Hslo, Is a Target of cGMP-dependent Protein Kinase Phosphorylation in VivoJournal of Biological Chemistry, 1998
- Smooth muscle cell expression of type I cyclic GMP-dependent protein kinase is suppressed by continuous exposure to nitrovasodilators, theophylline, cyclic GMP, and cyclic AMP.Journal of Clinical Investigation, 1997
- Protein Phosphatase 2A Is Essential for the Activation of Ca2+-activated K+ Currents by cGMP-dependent Protein Kinase in Tracheal Smooth Muscle and Chinese Hamster Ovary CellsJournal of Biological Chemistry, 1996
- Salt-resistant hypertension in mice lacking the guanylyl cyclase-A receptor for atrial natriuretic peptideNature, 1995
- Hypertension in mice lacking the gene for endothelial nitric oxide synthaseNature, 1995
- Modulation of Cyclic Guanosine Monophosphate Production during Escherichia coli Septic ShockBiochemical Medicine and Metabolic Biology, 1994
- Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscleNature, 1994
- Activation of protein kinase isozymes by cyclic nucleotide analogs used singly or in combinationEuropean Journal of Biochemistry, 1985