Normal Flux through ATP-Citrate Lyase or Fatty Acid Synthase Is Not Required for Glucose-stimulated Insulin Secretion
Open Access
- 1 October 2007
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 282 (43), 31592-31600
- https://doi.org/10.1074/jbc.m706080200
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- The inhibitors of protein acylation, cerulenin and tunicamycin, increase voltage-dependent Ca2+ currents in the insulin-secreting INS 832/13 cellBiochemical Pharmacology, 2007
- GPR40 Is Necessary but Not Sufficient for Fatty Acid Stimulation of Insulin Secretion In VivoDiabetes, 2007
- Mitochondrial GTP Regulates Glucose-Stimulated Insulin SecretionCell Metabolism, 2007
- Use of analogs and inhibitors to study the functional significance of protein palmitoylationMethods, 2006
- Pharmacological regulation of insulin secretion in MIN6 cells through the fatty acid receptor GPR40: identification of agonist and antagonist small moleculesBritish Journal of Pharmacology, 2006
- Inhibition of insulin secretion by cerulenin might be due to impaired glucose metabolismDiabetes/Metabolism Research and Reviews, 2006
- Free fatty acids regulate insulin secretion from pancreatic β cells through GPR40Nature, 2003
- Structure−Activity Studies of Cerulenin Analogues as Protein Palmitoylation InhibitorsJournal of Medicinal Chemistry, 1999
- Quantifying the Carboxylation of Pyruvate in Pancreatic IsletsJournal of Biological Chemistry, 1996
- Hexose metabolism in pancreatic islets. Effect of (−)-hydroxycitrate upon fatty acid synthesis and insulin release in glucose-stimulated isletsBiochimie, 1991