Transforming growth factor-β induces osteoclast formation in the absence of RANKL
- 31 January 2004
- Vol. 34 (1), 57-64
- https://doi.org/10.1016/j.bone.2003.08.008
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Features of mono‐ and multinucleated bone resorbing cells of the zebrafish Danio rerio and their contribution to skeletal development, remodeling, and growthJournal of Morphology, 2001
- Tumor Necrosis Factor α-Induced Osteoclastogenesis Requires Tumor Necrosis Factor Receptor-Associated Factor 6Journal of Bone and Mineral Research, 2001
- Transforming growth factor-β1 (TGF-β) stimulates the osteoclast-forming potential of peripheral blood hematopoietic precursors in a lymphocyte-rich microenvironmentBone, 2001
- Regulation of osteoclastogenesis and RANK expression by TGF‐β1Journal of Cellular Biochemistry, 2001
- The Roles of Osteoprotegerin and Osteoprotegerin Ligand in the Paracrine Regulation of Bone ResorptionJournal of Bone and Mineral Research, 2000
- TGF-β Enhances Osteoclast Differentiation in Hematopoietic Cell Cultures Stimulated with RANKL and M-CSFBiochemical and Biophysical Research Communications, 1999
- Transforming Growth Factor-β1 Increases mRNA Levels of Osteoclastogenesis Inhibitory Factor in Osteoblastic/Stromal Cells and Inhibits the Survival of Murine Osteoclast-like CellsBiochemical and Biophysical Research Communications, 1998
- REGULATION OF IMMUNE RESPONSES BY TGF-βAnnual Review of Immunology, 1998
- Transforming growth factor β1 stimulates bone formation and resorption in an in-vivo model in rabbitsBone, 1995
- Modulation of Macrophage Function by Transforming Growth Factor β, Interleukin‐4, and Interleukin‐10aAnnals of the New York Academy of Sciences, 1993