Venous calcification within the abdominal cavity, with the exception of phlebolith formation, is an extremely rare condition. Roentgen evidence of its occurrence is not to be found in the literature, although arterial changes have been frequently described. The few reported instances of venous calcification do not include radiographic studies, the changes having been found at autopsy or surgery. The occurrence of venous sclerosis in Banti's syndrome is especially interesting in view of the evidence that portal bed obstruction is probably the etiologic agent. Because of these features and the definite and striking roentgen picture, the following case was thought to be worthy of presentation. Case History L. L. B., a 49-year-old white male, was admitted to the Medical Service of the Wadsworth General Hospital, July 14, 1946. From 1919 to 1929, he had severe and repeated gastric hemorrhages, for which he was frequently hospitalized. He had been told that he had an enlarged spleen. In 1929, at another hospital, a splenectomy was done. Records obtained from that hospital are essentially unrevealing. A clinical diagnosis of Banti's or Gaucher's disease was made but there was no pathological examination. Following splenectomy, the patient remained well until May 1946, when he had a severe gastrointestinal hemorrhage, vomiting bright red blood and passing tarry stools. This occurred while he was on his honeymoon, and he was taken to a local hospital, where he had several more severe hemorrhages. He was later treated with multiple transfusions and was told he had a bleeding duodenal ulcer, shown radiographically. Three weeks later, while en route to the West Coast, he had another severe hemorrhage and one week before admission he had still another. He was hospitalized twice, en route, because of recurrent hemorrhage. On admission, the patient appeared rather thin and very pale. The physical examination was essentially negative except for a left upper rectus scar, a liver which was palpable two finger-breadths below the costal margin on deep inspiration, and internal hemorrhoids. Occult blood was found in the stool. The erythrocyte count was 3,100,000 with 56 per cent hemoglobin. The platelet count, prothrombin time, and fragility tests were normal. The urine examination revealed three-plus albumin, numerous red and white blood cells, and a low specific gravity. The urea nitrogen was 31.8 mg. per cent. The patient had several hemorrhages while on the Medical Service, the erythrocyte count at one time falling to 1,300,000 with 26 per cent hemoglobin. He was treated with multiple transfusions and was seen in surgical consultation. It was felt that his condition was too poor for any major surgical procedure at this time and conservative therapy and repeated transfusions were therefore recommended. When he improved he was transferred to the Surgical Service.