Nicotine increases human polymorphonuclear leukocytes chemotactic response —a possible additional mechanism of lung injury in cigarette smokers
- 1 January 1990
- journal article
- research article
- Published by Elsevier in Experimental pathology
- Vol. 39 (1), 37-43
- https://doi.org/10.1016/s0232-1513(11)80218-5
Abstract
No abstract availableThis publication has 19 references indexed in Scilit:
- Cigarette smoke can activate the alternative pathway of complement in vitro by modifying the third component of complement.Journal of Clinical Investigation, 1985
- Nicotine is Chemotactic for Neutrophils and Enhances Neutrophil Responsiveness to Chemotactic PeptidesScience, 1984
- Evidence for a noncholinergic nicotine receptor on human phagocytic leukocytesMolecular and Cellular Biochemistry, 1982
- Chemotactic activity of elastin-derived peptides.Journal of Clinical Investigation, 1980
- Human Alveolar Macrophage-derived Chemotactic Factor for NeutrophilsJournal of Clinical Investigation, 1980
- Relation of nicotine yield of cigarettes to blood nicotine concentrations in smokers.BMJ, 1980
- The Extracellular Release of Granulocyte Collagenase and Elastase during Phagocytosis and Inflammatory ProcessesScandinavian Journal of Haematology, 1977
- Collagen-and collagen peptide-induced chemotaxis of human blood monocytes.The Journal of Experimental Medicine, 1976
- The structure-activity relations of synthetic peptides as chemotactic factors and inducers of lysosomal secretion for neutrophils.The Journal of Experimental Medicine, 1976
- Pathologic Changes in the Peripheral Airways of Young Cigarette SmokersNew England Journal of Medicine, 1974