PARP is activated at stalled forks to mediate Mre11-dependent replication restart and recombination
Top Cited Papers
- 23 July 2009
- journal article
- research article
- Published by Springer Nature in The EMBO Journal
- Vol. 28 (17), 2601-2615
- https://doi.org/10.1038/emboj.2009.206
Abstract
EMBO Press is an editorially independent publishing platform for the development of EMBO scientific publications.Keywords
This publication has 67 references indexed in Scilit:
- Mre11 Dimers Coordinate DNA End Bridging and Nuclease Processing in Double-Strand-Break RepairCell, 2008
- Mre11 Nuclease Activity Has Essential Roles in DNA Repair and Genomic Stability Distinct from ATM ActivationCell, 2008
- Claspin Promotes Normal Replication Fork Rates in Human CellsMolecular Biology of the Cell, 2008
- Human CtIP promotes DNA end resectionNature, 2007
- Poly(ADP-Ribose) Polymerase 1 Accelerates Single-Strand Break Repair in Concert with Poly(ADP-Ribose) GlycohydrolaseMolecular and Cellular Biology, 2007
- Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replicationNature, 2006
- Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpointsNature, 2006
- ATM and ATR promote Mre11 dependent restart of collapsed replication forks and prevent accumulation of DNA breaksThe EMBO Journal, 2006
- Replication Protein A and the Mre11·Rad50·Nbs1 Complex Co-localize and Interact at Sites of Stalled Replication ForksJournal of Biological Chemistry, 2004
- Role of poly(ADP-ribose) formation in DNA repairNature, 1992