Complement Genes Strongly Predict Recurrence and Graft Outcome in Adult Renal Transplant Recipients with Atypical Hemolytic and Uremic Syndrome
Top Cited Papers
Open Access
- 28 January 2013
- journal article
- research article
- Published by Elsevier in American Journal of Transplantation
- Vol. 13 (3), 663-675
- https://doi.org/10.1111/ajt.12077
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- Common genetic variants in complement genes other than CFH, CD46 and the CFHRs are not associated with aHUSMolecular Immunology, 2011
- Long-term renal function under plasma exchange in atypical hemolytic uremic syndromePediatric Nephrology, 2011
- Common polymorphisms in C3, factor B, and factor H collaborate to determine systemic complement activity and disease riskProceedings of the National Academy of Sciences, 2011
- Prevention of large-vessel stenoses in atypical hemolytic uremic syndrome associated with complement dysregulationPediatric Nephrology, 2010
- Recurrent Atypical Hemolytic Uremic Syndrome Associated With Factor I Mutation in a Living Related Renal Transplant RecipientAmerican Journal of Kidney Diseases, 2009
- Mutations in complement C3 predispose to development of atypical hemolytic uremic syndromeBlood, 2008
- Spontaneous hemolytic uremic syndrome triggered by complement factor H lacking surface recognition domainsThe Journal of Experimental Medicine, 2007
- Gain-of-function mutations in complement factor B are associated with atypical hemolytic uremic syndromeProceedings of the National Academy of Sciences, 2007
- Modeling how CD46 deficiency predisposes to atypical hemolytic uremic syndromeMolecular Immunology, 2006
- Genetics of HUS: the impact of MCP, CFH, and IF mutations on clinical presentation, response to treatment, and outcomeBlood, 2006