Bax Does Not Directly Participate in the Ca2+-induced Permeability Transition of Isolated Mitochondria
Open Access
- 3 September 2004
- journal article
- Published by Elsevier
- Vol. 279 (36), 37415-37422
- https://doi.org/10.1074/jbc.m314093200
Abstract
No abstract availableKeywords
This publication has 72 references indexed in Scilit:
- BAX and BAK Regulation of Endoplasmic Reticulum Ca 2+ : A Control Point for ApoptosisScience, 2003
- Association of Bax and Bak Homo-oligomers in MitochondriaJournal of Biological Chemistry, 2003
- β2-microglobulin induces apoptosis in HL-60 human leukemia cell line and its multidrug resistant variants overexpressing MRP1 but lacking Bax or overexpressing P-glycoproteinOncogene, 2001
- The Destabilization of Lipid Membranes Induced by the C-terminal Fragment of Caspase 8-cleaved Bid Is Inhibited by the N-terminal FragmentJournal of Biological Chemistry, 2000
- Bax and Adenine Nucleotide Translocator Cooperate in the Mitochondrial Control of ApoptosisScience, 1998
- Mitochondrial control of apoptosisImmunology Today, 1997
- Mitochondria and programmed cell death: back to the futureFEBS Letters, 1996
- The mitochondrial permeability transitionBiochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1995
- Distribution of control of oxidative phosphorylation in mitochondria oxidizing NAD-linked substratesBiochimica et Biophysica Acta (BBA) - Bioenergetics, 1991
- Membrane potential of mitochondria measured with an electrode sensitive to tetraphenyl phosphonium and relationship between proton electrochemical potential and phosphorylation potential in steady stateThe Journal of Membrane Biology, 1979