Sensitization for death receptor- or drug-induced apoptosis by re-expression of caspase-8 through demethylation or gene transfer
Top Cited Papers
- 13 September 2001
- journal article
- Published by Springer Nature in Oncogene
- Vol. 20 (41), 5865-5877
- https://doi.org/10.1038/sj.onc.1204750
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 50 references indexed in Scilit:
- TRAIL receptor-2 signals apoptosis through FADD and caspase-8Nature Cell Biology, 2000
- IAP family proteins---suppressors of apoptosisGenes & Development, 1999
- Structure and chromosome localization of the human CASP8 geneGene, 1999
- Activation of the CD95 (APO-1/Fas) pathway in drug- and γ-irradiation-induced apoptosis of brain tumor cellsCell Death & Differentiation, 1998
- TRAIL: a molecule with multiple receptors and control mechanismsCurrent Opinion in Immunology, 1998
- Deficient activation of the CD95 (APO-1/Fas) system in drug-resistant cellsLeukemia, 1997
- Cytotoxic drugs, programmed cell death, and the immune system: defining new roles in an old play.JNCI Journal of the National Cancer Institute, 1997
- In vitro activation of CPP32 and Mch3 by Mch4, a novel human apoptotic cysteine protease containing two FADD-like domains.Proceedings of the National Academy of Sciences, 1996
- Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cellsNature Medicine, 1996
- FADD/MORT1 Is a Common Mediator of CD95 (Fas/APO-1) and Tumor Necrosis Factor Receptor-induced ApoptosisJournal of Biological Chemistry, 1996