The Role of Interleukin-4 and Interleukin-12 in the Progression of Atherosclerosis in Apolipoprotein E-Deficient Mice
Open Access
- 1 September 2003
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 163 (3), 1117-1125
- https://doi.org/10.1016/s0002-9440(10)63471-2
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- Immune Mechanisms in AtherosclerosisArteriosclerosis, Thrombosis, and Vascular Biology, 2001
- Regulation of vascular cell adhesion molecule-1 expression by IL-4 and TNF-alpha in cultured endothelial cells.JCI Insight, 1995
- Lymphocyte responses and cytokinesCell, 1994
- Disruption of the murine IL-4 gene blocks Th2 cytokine responsesNature, 1993
- Spontaneous Hypercholesterolemia and Arterial Lesions in Mice Lacking Apolipoprotein EScience, 1992
- Generation of mice carrying a mutant apolipoprotein E gene inactivated by gene targeting in embryonic stem cells.Proceedings of the National Academy of Sciences, 1992
- Natural killer cell stimulatory factor (NKSF) or interleukin-12 is a key regulator of immune response and inflammationProgress in Growth Factor Research, 1992
- Generation and Analysis of Interleukin-4 Deficient MiceScience, 1991
- Immunology of atherosclerosis: Cellular composition and major histocompatibility complex class II antigen expression in aortic intima, fatty streaks, and atherosclerotic plaques in young and aged human specimensClinical Immunology and Immunopathology, 1990
- Localization of T lymphocytes and macrophages in fibrous and complicated human atherosclerotic plaquesAtherosclerosis, 1988