Cerebrovascular control mechanisms, drug effects and brain ischemia as well as an insight into new techniques were discussed. With regard to autonomic nervous control mechanisms of cerebral blood flow, it was realized that steady state measurements may not detect short-term flow changes, e.g., caused by sympathetic stimulation. The autonomic nervous system may produce rapid flow adjustments in anticipation of subsequent steady state hemodynamic changes dictated by variations in metabolic rate. A new type of clinical neurophysiology based on 3-Dimensional studies of the cerebral blood flow and metabolism may highly increase understanding of both normal and abnormal (including epileptic) EEG patterns.