Phospholipase A2-derived lysophosphatidylcholine triggers Ca2+ entry in dystrophic skeletal muscle fibers
- 1 January 2010
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 391 (1), 401-406
- https://doi.org/10.1016/j.bbrc.2009.11.070
Abstract
No abstract availableKeywords
Funding Information
- Swiss National Science Foundation (31-109981.05, 31-122548.08)
- Swiss Foundation for Research on Muscular Diseases
- Association Française contre les Myopathies
This publication has 31 references indexed in Scilit:
- Regulation of capacitative calcium entries by α1‐syntrophin: association of TRPC1 with dystrophin complex and the PDZ domain of α1‐syntrophinThe FASEB Journal, 2007
- Regulation of store‐operated calcium entries and mitochondrial uptake by minidystrophin expression in cultured myotubesThe FASEB Journal, 2005
- Mechanisms of stretch‐induced muscle damage in normal and dystrophic muscle: role of ionic changesThe Journal of Physiology, 2005
- New aspects of calcium signaling in skeletal muscle cells: implications in Duchenne muscular dystrophyBiochimica et Biophysica Acta (BBA) - Proteins and Proteomics, 2002
- Involvement of TRPC in the abnormal calcium influx observed in dystrophic (mdx) mouse skeletal muscle fibersThe Journal of cell biology, 2002
- Long‐term study of Ca2+ homeostasis and of survival in collagenase‐isolated muscle fibres from normal and mdx miceThe Journal of Physiology, 2002
- Function and Genetics of Dystrophin and Dystrophin-Related Proteins in MusclePhysiological Reviews, 2002
- Calcium Influx through Calcium Leak Channels Is Responsible for the Elevated Levels of Calcium-dependent Proteolysis in Dystrophic MyotubesJournal of Biological Chemistry, 2000
- Calcium homeostasis in dystrophic muscleTrends in Pharmacological Sciences, 1999
- Increased calcium entry into dystrophin‐deficient muscle fibres of MDX and ADR‐MDX mice is reduced by ion channel blockersThe Journal of Physiology, 1999