Chronic administration of the GABA-transaminase inhibitor ethanolamine O-sulphate leads to up-regulation of GABA binding sites
- 1 February 1984
- journal article
- research article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 33 (3), 387-393
- https://doi.org/10.1016/0006-2952(84)90230-2
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Cerebral Glutamic Acid Decarboxylase Activity and γ‐Aminobutyric Acid Concentrations in Mice Susceptible or Resistant to Audiogenic SeizuresJournal of Neurochemistry, 1982
- γ‐Aminobutyric Acid and Benzodiazepine Binding Sites in Audiogenic Seizure‐Susceptible MiceJournal of Neurochemistry, 1982
- A Regional Study of 4‐Aminobutyrate Metabolism and Amino Acid Levels in Rat Brain Following Chronic Oral Administration of Ethanolamine O‐SulphateJournal of Neurochemistry, 1982
- GABA‐Benzodiazepine‐Barbiturate Receptor InteractionsJournal of Neurochemistry, 1981
- Receptor Adaptations to Centrally Acting DrugsAnnual Review of Pharmacology and Toxicology, 1981
- Anorexic effects of ethanolamine-O-sulfate and muscimol in the rat: Evidence that GABA inhibits ingestive behaviorLife Sciences, 1980
- Dopamine Receptor Supersensitivity in the Corpus Striatum Following Chronic Elevation of Brain γ‐Aminobutyric AcidJournal of Neurochemistry, 1980
- γ‐VINYL GABA: EFFECTS OF CHRONIC ADMINISTRATION ON THE METABOLISM OF GABA AND OTHER AMINO COMPOUNDS IN RAT BRAINJournal of Neurochemistry, 1979
- Molecular Mechanisms Mediating the Action of Diazepam on GABA ReceptorsThe British Journal of Psychiatry, 1978
- REDUCTION OF LEVEL OF L‐GLUTAMIC ACID DECARBOXYLASE BY γ‐AMINOBUTYRIC ACID IN MOUSE BRAIN1Journal of Neurochemistry, 1970