Orange juice neutralizes the proinflammatory effect of a high-fat, high-carbohydrate meal and prevents endotoxin increase and Toll-like receptor expression
- 1 April 2010
- journal article
- clinical trial
- Published by Elsevier in The American Journal of Clinical Nutrition
- Vol. 91 (4), 940-949
- https://doi.org/10.3945/ajcn.2009.28584
Abstract
Background: The intake of glucose or a high-fat, high-carbohydrate (HFHC) meal, but not orange juice, induces an increase in inflammation and oxidatiKeywords
This publication has 46 references indexed in Scilit:
- Increase in Plasma Endotoxin Concentrations and the Expression of Toll-Like Receptors and Suppressor of Cytokine Signaling-3 in Mononuclear Cells After a High-Fat, High-Carbohydrate MealDiabetes Care, 2009
- Chylomicrons promote intestinal absorption of lipopolysaccharidesJournal of Lipid Research, 2009
- Naringenin has anti‐inflammatory properties in macrophage and ex vivo human whole‐blood modelsJournal of Periodontal Research, 2008
- Fasting Compared With Nonfasting Triglycerides and Risk of Cardiovascular Events in WomenJAMA, 2007
- Metabolic Endotoxemia Initiates Obesity and Insulin ResistanceDiabetes, 2007
- The immunomodulation of endotoxin-induced acute lung injury by hesperidin in vivo and in vitroLife Sciences, 2007
- TLR4 links innate immunity and fatty acid–induced insulin resistanceJournal of Clinical Investigation, 2006
- Central role of endogenous Toll-like receptor-2 activation in regulating inflammation, reactive oxygen species production, and subsequent neointimal formation after vascular injuryBiochemical and Biophysical Research Communications, 2006
- Potential Role of Endotoxin as a Proinflammatory Mediator of AtherosclerosisArteriosclerosis, Thrombosis, and Vascular Biology, 2004
- Influence of Industrial Processing on Orange Juice Flavanone Solubility and Transformation to Chalcones under Gastrointestinal ConditionsJournal of Agricultural and Food Chemistry, 2003