The adenylate cyclase rebound response to naloxone in the NG108-15 cells
- 1 August 1986
- journal article
- research article
- Published by Elsevier in Neuropharmacology
- Vol. 25 (8), 833-837
- https://doi.org/10.1016/0028-3908(86)90007-9
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Cyclic AMP-dependent protein kinase closes the serotonin-sensitive K+channels of Aplysia sensory neurones in cell-free membrane patchesNature, 1985
- The effect of tolerance on opiate dependence as measured by the adenylate cyclase rebound response to naloxone in the NG108-15 model systemNeuropeptides, 1984
- How do opiates act?Trends in Neurosciences, 1984
- How do opiates inhibit neurotransmitter release?Trends in Neurosciences, 1983
- Modulation of adenylate cyclase activity by a cytosolic factor following chronic opiate exposure in neuroblastoma x glioma NG108-15 hybrid cellsLife Sciences, 1983
- Failure of opiates to increase the hydrolysis of GTP in neuroblastoma-glioma 108-15 cellsLife Sciences, 1983
- The role of protein phosphorylation in neural and hormonal control of cellular activityNature, 1982
- Serotonin induces a voltage-sensitive calcium current in neurons of Aplysia californica.Journal of Neurophysiology, 1980
- Phosphorylated Proteins as Physiological EffectorsScience, 1978
- Rates of onset and offset of action of narcotic analgesics in isolated preparationsEuropean Journal of Pharmacology, 1975