Cellular basis for long QT, transmural dispersion of repolarization, and torsade de pointes in the long QT syndrome
- 1 January 1999
- journal article
- review article
- Published by Elsevier in Journal of Electrocardiology
- Vol. 32, 177-184
- https://doi.org/10.1016/s0022-0736(99)90077-8
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Coassembly of KVLQT1 and minK (IsK) proteins to form cardiac IKS potassium channelNature, 1996
- Multiple Mechanisms in the Long-QT SyndromeCirculation, 1996
- Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmiasNature Genetics, 1996
- A mechanistic link between an inherited and an acquird cardiac arrthytmia: HERG encodes the IKr potassium channelCell, 1995
- SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndromeCell, 1995
- A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndromeCell, 1995
- Diagnostic criteria for the long QT syndrome. An update.Circulation, 1993
- Early afterdepolarizations induced by isoproterenol in patients with congenital long QT syndrome.Circulation, 1991
- The long QT syndrome: a prospective international study.Circulation, 1985
- The long Q-T syndromeAmerican Heart Journal, 1975