TAK1 Is a Master Regulator of Epidermal Homeostasis Involving Skin Inflammation and Apoptosis
Open Access
- 1 July 2006
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 281 (28), 19610-19617
- https://doi.org/10.1074/jbc.m603384200
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Skin lesion development in a mouse model of incontinentia pigmenti is triggered by NEMO deficiency in epidermal keratinocytes and requires TNF signalingHuman Molecular Genetics, 2006
- TAK1, but not TAB1 or TAB2, plays an essential role in multiple signaling pathways in vivoGenes & Development, 2005
- Negative regulation of Toll-like receptor-mediated immune responsesNature Reviews Immunology, 2005
- CDK4 regulation by TNFR1 and JNK is required for NF-κB–mediated epidermal growth controlThe Journal of cell biology, 2005
- Toll-like receptor signallingNature Reviews Immunology, 2004
- Characterization of the Progressive Skin Disease and Inflammatory Cell Infiltrate in Mice with Inhibited NF-κB SignalingJournal of Investigative Dermatology, 2004
- Signalling pathways of the TNF superfamily: a double-edged swordNature Reviews Immunology, 2003
- TAK1 is Critical for IκB Kinase-mediated Activation of the NF-κB PathwayJournal of Molecular Biology, 2003
- Getting under the skin of epidermal morphogenesisNature Reviews Genetics, 2002
- NEMO/IKKγ-Deficient Mice Model Incontinentia PigmentiMolecular Cell, 2000