Susceptibility of brain to AMPA induced excitotoxicity transiently peaks during early postnatal development
Open Access
- 26 June 1992
- journal article
- Published by Elsevier in Brain Research
- Vol. 583 (1-2), 54-70
- https://doi.org/10.1016/s0006-8993(10)80009-5
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- In Vitro and In Vivo Pharmacology of trans‐ and cis‐(±)‐1‐Amino‐1,3‐Cyclopentanedicarboxylic Acid: Dissociation of Metabotropic and Ionotropic Excitatory Amino Acid Receptor EffectsJournal of Neurochemistry, 1991
- Excitotoxic Brain Injury Suppresses Striatal High‐Affinity Glutamate Uptake in Perinatal RatsJournal of Neurochemistry, 1991
- Pharmacology of N‐methyl‐D‐aspartate‐induced brain injury in an in vivo perinatal rat modelSynapse, 1990
- Quantitative assessment of neuroprotection against NMDA-induced brain injuryExperimental Neurology, 1989
- Ketamine and MK-801 prevent degeneration of thalamic neurons induced by focal cortical seizuresExperimental Neurology, 1989
- Glutamate neurotoxicity and diseases of the nervous systemNeuron, 1988
- Agonists and antagonists for excitatory amino acid receptorsTrends in Neurosciences, 1987
- Excitotoxity and the NMDA receptorTrends in Neurosciences, 1987
- Kainate and glutamate neurotoxicity in dependence on the postnatal development with special reference to hippocampal neuronsDevelopmental Brain Research, 1984
- Effect of anticonvulsant drugs on kainic acid-induced epileptiform activityExperimental Neurology, 1982