Physiological Expression of the Gene for PrP-Like Protein, PrPLP/Dpl, by Brain Endothelial Cells and its Ectopic Expression in Neurons of PrP-Deficient Mice Ataxic Due to Purkinje Cell Degeneration
Open Access
- 30 November 2000
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 157 (5), 1447-1452
- https://doi.org/10.1016/s0002-9440(10)64782-7
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- PrP's Double Causes TroubleScience, 1999
- Ataxia in prion protein (PrP)-deficient mice is associated with upregulation of the novel PrP-like protein doppelJournal of Molecular Biology, 1999
- Immunoultrastructural expression of intercellular adhesion molecule-1 in endothelial cell vesiculotubular structures and vesiculovacuolar organelles in blood-brain barrier development and injuryCell and tissue research, 1999
- The human 37-kDa laminin receptor precursor interacts with the prion protein in eukaryotic cellsNature Medicine, 1997
- Loss of cerebellar Purkinje cells in aged mice homozygous for a disrupted PrP geneNature, 1996
- Role of microglia and host prion protein in neurotoxicity of a prion protein fragmentNature, 1996
- Normal development and behaviour of mice lacking the neuronal cell-surface PrP proteinNature, 1992
- Characterization of Endothelial Cell Transport in the Developing Mouse Blood-Brain BarrierDevelopmental Neuroscience, 1986
- Angiogenesis in developing rat brain: An in vivo and in vitro studyDevelopmental Brain Research, 1985
- Factor VIII related antigen as an endothelial cell marker in benign and malignant diseasesVirchows Archiv, 1981