Molecular memory by reversible translocation of calcium/calmodulin-dependent protein kinase II
- 1 September 2000
- journal article
- research article
- Published by Springer Nature in Nature Neuroscience
- Vol. 3 (9), 881-886
- https://doi.org/10.1038/78783
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Control of Memory Formation Through Regulated Expression of a CaMKII TransgeneScience, 1996
- Calcium/calmodulin-dependent kinase II and long-term potentiation enhance synaptic transmission by the same mechanism.Proceedings of the National Academy of Sciences, 1995
- The Multifunctional Calcium/Calmodulin-Dependent Protein Kinase: From Form to FunctionAnnual Review of Physiology, 1995
- CaMKII regulates the frequency-response function of hippocampal synapses for the production of both LTD and LTPCell, 1995
- Potentiated Transmission and Prevention of Further LTP by Increased CaMKII activity in Postsynaptic Hippocampal Slice NeuronsScience, 1994
- Deficient Hippocampal Long-Term Potentiation in α-Calcium-Calmodulin Kinase II Mutant MiceScience, 1992
- Impaired Spatial Learning in α-Calcium-Calmodulin Kinase II Mutant MiceScience, 1992
- Calmodulin Trapping by Calcium-Calmodulin-Dependent Protein KinaseScience, 1992
- Persistent protein kinase activity underlying long-term potentiationNature, 1988
- Regional distribution of type II Ca2+/calmodulin-dependent protein kinase in rat brainJournal of Neuroscience, 1985