• 1 January 1978
    • journal article
    • research article
    • Vol. 38 (3), 494-497
Abstract
Wounding by incision was a promoting stimulus in mouse skin previously initiated with 7,12-dimethylbenz(a)anthracene. Skin massage elicited a marked proliferative response in skin but was not a promoting stimulus. Wounding mouse skin, by multiple scalpel incisions or stripping with silicon carbide paper, led to a marked induction of ornithine decarboxylase activity. In both instances activity was maximal between 20 and 26 h after wounding, with a secondary rise at 72 h. Skin massage did not lead to a detectable increase in ornithine decarboxylase activity over the same time period.