Src kinase contributes to the metastatic spread of carcinoma cells
- 4 April 2002
- journal article
- research article
- Published by Springer Nature in Oncogene
- Vol. 21 (15), 2347-2356
- https://doi.org/10.1038/sj.onc.1205298
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 56 references indexed in Scilit:
- Growth defects induced by perturbation of β1‐integrin function in the mammary gland epithelium result from a lack of MAPK activation via the Shc and Akt pathwaysEMBO Reports, 2001
- The catalytic activity of Src is dispensable for translocation to focal adhesions but controls the turnover of these structures during cell motilityThe EMBO Journal, 1998
- Src and Ras are involved in separate pathways in epithelial cell scatteringThe EMBO Journal, 1997
- Epithelial-Mesenchymal Transitions in Cancer ProgressionCells Tissues Organs, 1996
- Myc but not Fos rescue of PDGF signalling block caused by kinase-inactive SrcNature, 1995
- Epithelium-Mesenchyme Transition during Neural Crest DevelopmentCells Tissues Organs, 1995
- Oncogenes and the strategy of growth factorsCell, 1994
- Redistribution of activated pp60c-src to integrin-dependent cytoskeletal complexes in thrombin-stimulated platelets.Molecular and Cellular Biology, 1993
- Expression of transfected transforming growth factor alpha induces a motile fibroblast-like phenotype with extracellular matrix-degrading potential in a rat bladder carcinoma cell line.Cell Regulation, 1990
- Transformation by Oncogenes Encoding Protein Kinases Induces the Metastatic PhenotypeScience, 1987