Deletion of Decay-Accelerating Factor (CD55) Exacerbates Autoimmune Disease Development in MRL/lpr Mice
- 30 September 2002
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 161 (3), 1077-1086
- https://doi.org/10.1016/s0002-9440(10)64268-x
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- ComplementNew England Journal of Medicine, 2001
- Complement C4 Inhibits Systemic Autoimmunity through a Mechanism Independent of Complement Receptors Cr1 and Cr2The Journal of Experimental Medicine, 2000
- The central and multiple roles of B cells in lupus pathogenesisImmunological Reviews, 1999
- Disruption of the Cr2 Locus Results in a Reduction in B-1a Cells and in an Impaired B Cell Response to T-Dependent AntigenImmunity, 1996
- C3d of Complement as a Molecular Adjuvant: Bridging Innate and Acquired ImmunityScience, 1996
- Polygenic control of susceptibility to murine systemic lupus erythematosusImmunity, 1994
- The evolution of mouse and human complement C3-binding proteins: divergence of form but conservation of functionImmunology Today, 1992
- Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosisNature, 1992
- Lpr and gld: Single Gene Models of Systemic Autoimmunity and Lymphoproliferative DiseaseAnnual Review of Immunology, 1991
- Decay-accelerating Factor in Human Skin Is Associated with Elastic FibersJournal of Investigative Dermatology, 1988