A cell-autonomous requirement for Cip/Kip cyclin-kinase inhibitors in regulating neuronal cell cycle exit but not differentiation in the developing spinal cord
- 27 October 2006
- journal article
- Published by Elsevier in Developmental Biology
- Vol. 301 (1), 14-26
- https://doi.org/10.1016/j.ydbio.2006.10.035
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Overexpression of p27Kip1, Probability of Cell Cycle Exit, and Laminar Destination of Neocortical NeuronsCerebral Cortex, 2005
- Transduction of graded Hedgehog signaling by a combination of Gli2 and Gli3 activator functions in the developing spinal cordDevelopment, 2004
- Pax6andEngrailed 1Regulate Two Distinct Aspects of Renshaw Cell DevelopmentJournal of Neuroscience, 2004
- Neuronal specification in the spinal cord: inductive signals and transcriptional codesNature Reviews Genetics, 2000
- Expression of the p16INK4a tumor suppressor versus other INK4 family members during mouse development and agingOncogene, 1997
- A Syndrome of Multiorgan Hyperplasia with Features of Gigantism, Tumorigenesis, and Female Sterility in p27 -Deficient MiceCell, 1996
- Enhanced Growth of Mice Lacking the Cyclin-Dependent Kinase Inhibitor Function of p27Cell, 1996
- Mice Lacking p27 Display Increased Body Size, Multiple Organ Hyperplasia, Retinal Dysplasia, and Pituitary TumorsCell, 1996
- D-type cyclinsTrends in Biochemical Sciences, 1995
- The retinoblastoma protein and cell cycle regulationTrends in Genetics, 1992